Studies on alcaptonuria: 2. Investigations on a case of human alcaptonuria.

نویسندگان

  • A Neuberger
  • C Rimington
  • J M Wilson
چکیده

Baumann, E. (1892). Hoppe-Seyl. Z. 16, 268. Blix, G. (1932). Hoppe-Seyl. Z. 210, 87. Briggs, A. P. (1922). J. biol. Chem. 51, 451. Faraday, M. (1857). Philos. Tran8. 147, 145. Garrod, A. E. & Hurtley, W. H. (1905). J. Phy8iol. 33, 206. James, T. H. (1939). J. Amer. chem. Soc. 61, 648. Lieb, H. & Lanyar, F. (1929). Hoppe-Seyl. Z. 181, 199. Medes, G. (1932). Biochem. J. 26, 917. Metz, E. (1927). Biochem. Z. 190, 261. Metz, E. (1930). Hoppe-Seyl. Z. 193, 46. Neuberger, A., Rimington, C. & Wilson, J. M. (1947). Biochem. J. 4i, 438. Neuberger, A. & Webster, T. A. (1947). Biochem. J. 41, 449. Sealock, R. R. & Silberstein, H. E. (1940). J. biol. Chem. 135, 251. Snell, J. M. & Weissberger, A. (1939). J. Amer. chem. Soc. 61, 450. Voigt, H. & Heumann, J. (1928). Z. anorg. Chem. 169, 140. Wieland, H. (1910). Ber. dt8ch. chem. Gme. 43, 710.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

A case of alcaptonuria with fatal cardiovascular disturbance.

A case of alcaptonuria combined with aortic insufficiency was found in a 28-year-old male. The patient was palpitating at admission. The daily excretion of homogentisic acid was 2.0-6.0 g. Electrocardiography indicated atrial fibrillation and left ventricular hypertrophy with a ST-T change and right axis deviation. Cartilage tissues in the knee-joints showed no pigmentation. Vertebral X-ray rev...

متن کامل

Problems in the laboratory diagnosis of alcaptonuria.

Two patients with urinary findings suggestive of alcaptonuria were observed. One was a two-year-old girl of Turkish descent, presenting with dark-stained diapers, black ear wax, and no other stated problem. The second was a 61-year-old North American Indian woman with long-standing rheumatoid arthritis, bluish sclerae, chronic renal failure, and dark urine. Diagnosis of alcaptonuria was confirm...

متن کامل

The nature of the defect in tyrosine metabolism in alcaptonuria.

Alcaptonuria is a rare, hereditary, metabolic disorder characterized by a defect in the oxidation of tyrosine (l-3). In this condition homogentisic acid, an intermediary product of tyrosine degradation in mammalian liver, is excreted in the urine. This disorder has been attributed to an abnormality of the enzyme system, homogentisic acid oxidase, but the exact nature of the abnormality has not ...

متن کامل

Observations on the oxidation of homogentisic acid in urine.

Homogentisic acid, which is considered to be an intermediate in the metabolism of tyrosine and phenylalanine, is excreted in a number of different conditions. The most widely studied of these, which is caused by a genetic inability to oxidize homogentisic acid, was termed alcaptonuria, owing to the characteristic darkening of the urine on standing. It has also been shown (Sealock & Silberstein,...

متن کامل

Xxxviii. Metabolic Studies in Phenylketonuria

F6LLING [1934] discovered that, in certain cases of mental deficiency, phenylpyruvic acid was excreted in the urine. His attention was drawn to the phenomenon by the characteristic colour produced by the addition of ferric chloride solution to the urines of these patients, and he identified the responsible substance as phenylpyruvic acid. Other cases have since been recognized [Penrose, 1935]. ...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:
  • The Biochemical journal

دوره 41 3  شماره 

صفحات  -

تاریخ انتشار 1947